Magnetonin Overview

What is MagnetoninTM?
Magnetonin is the name given to the polypeptide hormone that is presumed (at this point in time) to be responsible for the regulation of magnesium uptake, excretion and level control in blood and tissues.

What is the origin of Magnetonin concept?
The Magnetonin Concept has come about as a result of research being carried out as part of the Mg Project of IPRS, Inc.

What is the evidence for Magnetonin?
All other major electrolytes of the body are regulated by one or more regulatory mechanisms including but not limited to a hormone that when released elevates the level of the cation in the blood and in selected tissues/organs.  Given the importance of Mg levels in the blood and many different tissues, it seems highly probable that there is also a hormone to regulate it.

What will it do?
It should be active on promoting uptake from the gut, re-absorption from the kidneys and mobilization from bone.  It may also activate Mg-channels in muscle and neural tissues where Mg is especially vital to normal functioning. Likely targets include Mg transporter genes such as TRPM7.  Since Mg plays a vital role in over three hundred (300+) biochemical reactions and pathways, it is also likely that magnetonin will influence many normal and disease processes.

Where is it Produced?
If similar to calcitonin, it will likely be found in a more concentrated form in the parathyroid glands.  It may also be produced in less concentrated amounts in key target tissues.  In anadromous fish (like salmon), the target tissues are likely in the gills and ultimobranchial body, those tissues involved in electrolyte regulation.

Objectives
To identify the hormone, determine its structure and various analogs and to develop patentable commercial forms that may be licensed for commercial development.

How to Participate?
One of the possible ways to participate would be through a sponsorship program.  Another would be as research collaborator.

Links

www.magnetonin.com

www.mgportal.org

www.iprsinc.org

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© 2006 - 2024  IPRS, Inc.
Revised: February 16, 2024